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Jean Isnard

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4 papers
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4

YNIMG Journal 2025 Journal Article

Cardiac autonomic responses to cortical electrical stimulation: a SEEG study

  • Florian Chouchou
  • Hugo Soulier
  • Vincent Pichot
  • François Mauguière
  • Isabelle Faillenot
  • Marc Guénot
  • Marc Hermier
  • Julien Jung

Recent growing neuroimaging evidence support that a set of cortical regions - the central autonomic network - is involved in autonomic control, but its functional organization remains unclear. We studied the direct autonomic cardiac effects produced by 1500 direct cortical electrical stimulations in 43 patients with epilepsy (32.8 ± 8.6 years old, 19 females) undergoing intracerebral recordings during presurgical evaluation. The time course of RR interval (RRI) reactivity and its variability were studied. Nearly half (48.6 %, n = 729) of the cortical stimulations resulted in a cardiac response, divided almost equally between bradycardia (24.47 %) and tachycardia (24.13 %), with no difference between right and left stimulations. Bradycardia was marked by an increase in parasympathetic heart control (increase in HF power and decrease in LF/HF ratio), while tachycardia was marked by a predominance in sympathetic heart control (decrease in HF power and increase in LF/HF ratio). We individualized a main network, where evoked bradycardia and tachycardia were strong, consisting of amygdala, posterior insula, frontal mesial premotor/prefrontal cortex, and anterior cingulate. Other brain regions were also involved, but to a lesser degree, with regions mostly in the limbic system and neocortex (sensory-motor/premotor and lateral temporal regions). These results highlight a close relationship between cerebral cortex and heart. Two hierarchically ordered networks were identified. A 'core' autonomic network strongly involved in cardiovascular regulation, consistent with the classical definition of CAN in functional imaging. But also a more 'widespread' autonomic network, both consistent with a major role of the cortex in continuous autonomic cardiac adjustments to high level emotional, cognitive or sensorimotor cortical activities. This study establishes for the first time a functional mapping of cardiac responses evoked by cortical electrical stimulations, and evidenced hierarchically ordered networks that extends the classical model of CAN.

YNICL Journal 2017 Journal Article

The relationship between morphological lesion, magnetic source imaging, and intracranial stereo-electroencephalography in focal cortical dysplasia

  • Romain Bouet
  • François Mauguière
  • Sébastien Daligault
  • Jean Isnard
  • Marc Guenot
  • Olivier Bertrand
  • Julien Jung

Magnetoencephalography (MEG) is a useful non-invasive technique for presurgical evaluation of focal cortical dysplasia patients. We aimed at clarifying the precise spatial relationship between the spiking volume determined with MEG, the seizure onset zone and the lesional volume in patients with focal cortical dysplasia. We studied the spatial relationships between the MEG spiking volume determined with a recent analysis pipeline, the seizure-onset zone location determined with a quantitative index calculated from intracranial EEG signals ('Epileptogenicity Index') and the lesional volume delineated on brain MRI in 11 patients with Focal Cortical Dysplasia explored with Stereo-electroencephalography (SEEG). A significant correlation between the MEG spiking activity and the Epileptogenicity Index was found in 8/11 patients. 7/8 patients were operated upon and had good surgical outcome. For three patients, no correlation between Epileptogenicity Index and spiking activity was observed; only one of those three patients had good surgical outcome. The lesion was at least partially overlapping with the seizure-onset zone in 8/9 patients with a lesion clearly identifiable by MRI. However, 57% of the SEEG epileptogenic contacts were located outside of the lesional volume. Lastly 44% of the highly epileptogenic SEEG contacts were located within the spiking volume and 22% of them were located exclusively in the spiking volume and not in the lesion. For 7/9 patients with a lesion, < 50% of epileptogenic SEEG contacts were included within the lesion: for 5/7 patients MEG provided an added value for targeting the epileptogenic region through intracranial electrodes, while for two of seven patients MEG detected only a few extralesional epileptogenic contacts. Our study suggests that modeling of the spiking volume with MEG is a promising tool to localize non-invasively the seizure-onset zone in patients with focal cortical dysplasia. Combined with brain MRI, MEG modeling of the spiking volume contributes to delineate the spatial extent of the seizure-onset zone.